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The Alzheimer’s Disease-Associated Amyloid β-Protein Is an Antimicrobial Peptide, Soscia S. et al., PLoS ONE (2010) 5(3): e9505.

Amyloid-β peptide protects against microbial infection in mouse and worm models of Alzheimer’s disease, Kumar D. et al., Science Translational Medicine (2016) 8(340): 340ra72–340ra72.

Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer’s disease, Lambert J. et al., Nature Genetics (2013) 45(12): 1452–1458.

Corroboration of a Major Role for Herpes Simplex Virus Type 1 in Alzheimer’s Disease, Itzhaki R., Frontiers in Aging Neuroscience (2018) 10(Oct) 324.

Anti-herpetic Medications and Reduced Risk of Dementia in Patients with Herpes Simplex Virus Infections—a Nationwide, Population-Based Cohort Study in Taiwan, Tzeng N. et al., Neurotherapeutics (2018) 15(2): 417–429.

Herpes simplex virus infection causes cellular β-amyloid accumulation and secretase upregulation, Wozniak M., Itzhaki R., Shipley S., Dobson C., Neuroscience Letters (2007) 429(2–3): 95–100.

Antivirals reduce the formation of key Alzheimer’s disease molecules in cell cultures acutely infected with herpes simplex virus type 1, Wozniak M., Frost A., Preston C., Itzhaki R., PLoS ONE (2011) 6(10).

Herpes simplex virus type 1 DNA is located within Alzheimer’s disease amyloid plaques, Wozniak M., Mee A., Itzhaki R., Journal of Pathology (2009) 217(1): 131–138.

Porphyromonas gingivalis in Alzheimer’s disease brains: Evidence for disease causation and treatment with small-molecule inhibitors, Dominy S. et al., Science Advances (2019) 5(1).

Periodontal disease and incident dementia: The Atherosclerosis Risk in Communities Study (ARIC), Demmer R. et al., Neurology (2020) 95(12): e1660–e1671.

Association between periodontal pathogens and systemic disease, Bui F. et al., 2019 Feb; 42(1): 27–35.

Chlamydophila pneumoniae and the etiology of late-onset alzheimer’s disease, Balin B et al., Alzheimers Dis, 2008 May; 13(4): 371–80.

Identification and localization of Chlamydia pneumoniae in the Alzheimer’s brain, Balin B. et al., Medical Microbiology and Immunology (1998) 187(1): 23–42.

Different Brain Regions are Infected with Fungi in Alzheimer’s Disease, Pisa D., Alonso R., Rábano A., Rodal I., Carrasco L., Scientific Reports (2015) 5(1): 1–13.

Microglia and amyloid precursor protein coordinate control of transient Candida cerebritis with memory deficits, Wu Y., Nat Commun 10, 58 (2019).

Amyloid beta and the longest-lived rodent: The naked mole-rat as a model for natural protection from alzheimer’s disease, Edrey Y., Medina D., Gaczynska M., Osmulski P., Oddo S., Caccamo A., Buffenstein R., Neurobiology of Aging (2013) 34(10): 2352–2360.

The Involution of the Ageing Human Thymic Epithelium is Independent of Puberty: A Morphometric Study, Steinmann G., Klaus B., Müller‐Hermelink H., Scandinavian Journal of Immunology (1985) 22(5): 563–575.